teratogenic effects of exercise and pregnancy outcome in mice
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teratogenic effects of exercise and pregnancy outcome in mice

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Published .
Written in English


  • Pregnancy -- Exercises.,
  • Teratogenic agents.

Book details:

Edition Notes

Statementby William Henry Boehnke.
The Physical Object
Paginationx, 63 leaves, bound :
Number of Pages63
ID Numbers
Open LibraryOL16586562M

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Investigation of the teratogenic effects of exercise on pregnancy outcome in mice. Boehnke WH, Chernoff GF, Finnell RH. Swiss Webster female mice were arbitrarily assigned to a heavily exercised group (HE), a moderately exercised group (ME), or a sedentary group (S).Cited by: 5. Pain can have negative, physiological and psychological impacts on pregnancy. Pregnant women are fearful of using pain medication because of teratogenic effects. In this study, we evaluated whether.   The dual stresses of pregnancy and exercise may create conflicting physiological demands that could adversely affect pregnancy outcome. Specifically, redistribution of uterine blood flow and subsequent fetal hypoxia, hyperthermia and the risk of teratogenic effects, decreased carbohydrate availability for the fetus, and increased uterine contractility with a possible increase in Cited by: teratogenic effects of overheating. Pregnant women should avoid exercise that involves the risk of abdominal trauma, falls or excessive joint stress, as in contact sports and vigorous racquet sports. In the absence of any obstetric or medical complications, most women can maintain a regular exercise regimen during pregnancy.

Introduction. The benefits of exercise training (ExT) have been widely studied in healthy individuals, as well as in patients with cardiovascular diseases (CVD), such as hypertension and atherosclerosis. 1 In addition, it has been shown to improve normal pregnancy outcomes. 2 Preeclampsia, a gestational disease, complicates ≈5% to 10% of pregnancies and is the leading cause of maternal and. This multi-author work deals with the practical aspects of teratogens - chemicals which cause birth defects. It is designed for use as a unique guide to these chemicals in which one can find all relevant information. The issues covered include: how to obtain information about the teratogenic potential of chemicals; teratogenic chemicals in undergraduate chemistry laboratories; safe handling of. stages of pregnancy (early, mid, and late). The results showed that not associated with adverse outcomes. Analysis, however showed tendencies toward adverse effects of alcohol consumption. One or more alcoholic drinks per day consumed during early pregnancy had a correlation to birth weight. Teratogenic agents cause approximately 7% of congenital malformations. A teratogenic agent is a chemical, infectious agent, physical condition, or deficiency that, on fetal exposure, can alter fetal morphology or subsequent function. Teratogenicity depends upon .

  There are no human data for early pregnancy use and such postulation needs further confirmation. Ribavirin was also associated with significant teratogenic effects in animal studies, Patients who decide to use ribavirin in early pregnancy should be given the option for termination of pregnancy. Effects of teratogens during this period of developmental often times results in an “all or none effect.” That is, the effect of the teratogen, if it is to have any effect, will be so profound as to cause a spontaneous abortion. Some examples of teratogens known to cause human malformations are listed in the table below (Table ). Teratogenic effects of thalidomide are well described in several animal models. Pregnant cats have tolerated doses of mg kg −1 day −1 of thalidomide with no fetal toxicity evident in offspring. Many rat strains have had no teratogenic effects seen at doses of mg kg −1 day −1 during pregnancy. ARTICLE Investigation of possible teratogenic effects in the offspring of mice exposed to methylphenidate during pregnancy Gabriel de Araújo Costa a, Talita Cristina Galvão a, André Demambre Bacchi b, Estefânia Gastaldello Moreira b, Maria José Sparça Salles a,* a Department of Biology, State University of Londrina (UEL), , Londrina, Parana, Brazil; b Department of.